COLOR-VISION DEFICIENCY
When I get dressed, my wife
checks that the colors I choose match, says Michael. At breakfast she selects a
piece of fruit for me because I can’t see if the fruit is ripe. At work I can’t
always see where to click on the computer screen, since items are often
distinguished by color. When I’m driving, red and green traffic lights appear
the same to me, so I observe whether the illuminated light is on top or on the
bottom. Horizontal lights, however, can present a problem.
HOW DO WE SEE COLORS
Light from object passes through
the CORNEA and the LENS and is focused on the RETINA. THE RETINA contains CONE
CELLS and ROD CELLS. Together they give the full range of vision. THE CONE
CELLS are sensitive to RED,
GREEN, OR BLUE light. The OPTIC NERVE
carries visual impulses to the brain. The image is inverted but corrected later
by the brain.
TEST FOR COLOR-VISION DEFICIENCY
Tests to discover the type and
degree of color-vision deficiency that a person has often employ patterns of
dots in various hues and shades. The widely used Ishihara test consists of up
to 38 different patterns.
For example, when viewing one of the test
patterns in daylight, a person with normal vision should see the numbers 42 and
74, while someone having a red-green deficiency –the most common –may see no
numbers at the top and 21 at the bottom. If testing reveals a defect, an eye
doctor may recommend further tests to determine whether it was inherited or has
some other cause.
WHY MAINLY MALES?
Inherited color deficiencies are
carried on the X chromosome. Women have two X chromosomes, while men have one X
and one Y. Thus, if a woman inherits a visual defect in an X chromosome, the
normal gene in the other chromosome will likely override it, and her vision
will be fine. But a man who inherits a defect on his X chromosome has no other
X chromosome to fall back on.
Michael has color-vision deficiency, also
called color deficiency or color blindness. He inherited a genetic flaw that
causes a defect in the retina –the light-sensitive inner lining of the eyes.
Michael shares this condition with about 1 of every 12 males of European
ancestry and about 1 of every 200 females.
Like the vast majority of
sufferers, Michael can see different colors –he does not see only black and
white. But some colors do not look the same to him as they do to people with
normal vision.
In the human eye, the retina
normally contains three kinds of CONE-SHAPED color-sensitive cells. Each kind
is tuned to the wavelength of a different primary color of light –blue, green,
or red. Light of different wavelengths triggers the corresponding cones, which
signal the brain and enable one to perceive colors.
In people with color-vision
deficiency, however, the sensitivity of the cones to one or more colors is weak
or shifted in wavelength, so that their response to color is altered. Most
sufferers have difficulty distinguished between yellow, green, orange, red, and
brown.
This effect can make it hard to
see green mold on brown bread or on yellow cheese or to distinguish a blue-eyed
blonde from a green-eyed redhead. If a person’s red-sensitive cones are very
weak, a red rose appears black. Very few sufferers cannot see blue.
Color-vision deficiencies can be
found in all racial groups, but it is most common among Caucasians. Many
animals can discern colors, although their color vision differs from ours.
Dogs, for example, have only two kinds of cones in their retinas –one for blue
and the other for a hue between red and green. Some birds, on the other hand,
have four kinds of cones and can detect ultraviolet light, which is beyond the
human range.
COLOR-VISION DEFICIENCY AND CHILDREN
Defects in color vision are
usually inherited and present at birth, and children with the condition often
learn unconsciously to compensate. For instance, even if they cannot see the
difference between certain hues, they may perceive differences in contrast and
brightness and associate these variations with the names given to the colors.
They may also learn to identify
objects by surface patterns and textures instead of by color. In fact, many
young people remain unaware of their disability throughout childhood.
Because schools often use color-coded teaching
tools, especially in the early grades, parents and teachers may mistakenly
think that a child has a learning disability when in fact, he may have a
color-vision deficiency.
One teacher even punished a
five-year old boy for painting a picture that had pink clouds, green people,
and trees with brown leaves. To a child with color-vision deficiency, these
colors may seem perfectly normal. For good reason, therefore, some authorities
recommend routine color-vision testing in early childhood.
Color-vision deficiency can
sometimes be caused by disease if you notice changes in your color vision later
in life. Although there is no known cure for this condition, it neither worsens
with age nor increases the risk for other defects in vision. Still,
color-vision deficiency is a disability that can be frustrating.
I understand how scarlet can
differ from crimson because I know that the smell of an orange is not the smell
of a grapefruit. I can also conceive that color have shades and guess what
shades are. In smell and taste there are varieties not broad enough to be
fundamental; so I call them shades…..
The force of association drives
me to say that white is exalted and pure, green is exuberant, red is suggests
love or shame or strength. Without the color or its equivalent, life to me
would be dark, barren, a vast blackness.
Thus through an inner law of
completeness my thoughts are not permitted to remain colorless. It strains my
mind to separate color and sound from objects. Since my education began I have
always had things described to me with their colors and sounds, by one with
keen senses and a fine feeling for the significant.
Therefore, I habitually think of
things as colored and resonant. Habit accounts for part. The soul sense account
for another part. The brain with its five-sensed construction asserts its right
and accounts for the rest.
Inclusive of all, the unity of
the world demands that color be kept in it whether I have cognizance of it or
not. Rather than be shut out, I take part in it by discussing it, happy in the
happiness of those near to me who gaze at the lovely hues of the sunset or the
rainbow.
THE RETINA
The human eye contains a RETINA
–a membrane with approximately 120 million cells called PHOTORECEPTORS, which
absorb light rays and convert them into electric signals. Your brain interprets
these signals as visual images. Evolutionists have contended that where the
retina is placed in the eyes of vertebrates, creatures with a backbone, proves
that the eye had no designer.
The retina of VERTEBRATES is
inverted, placing the photoreceptors at the back of the retina. To reach them,
light must pass through several layers of cells. According to evolutionary
biologist Kenneth Miller, this arrangement SCATTERS the light, making our
vision less detailed than it might be.
Evolutionists thus claim that the
inverted retina is evidence of poor design –really, no design. One scientist
even described it as a functionally stupid upside-down orientation. However,
further research reveals that the photoreceptors of the inverted retina are
ideally placed next to the pigment epithelium –a cell layer that provides
oxygen and nutrients vital to keen sight. If the pigment epithelium tissue were
placed IN FRONT of the retina, sight would be seriously compromised.
The inverted retina is especially
advantageous for vertebrates with small eyes. Between the lens of the eye and
the photoreceptors, there must be a certain distance to get a sharp image.
Having this space filled with nerve cells means an important saving of space
for vertebrates.
Additionally, with the nerve
cells of the retina tightly packed and close to the photoreceptors, analysis of
visual information is fast and reliable.
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